VOMITING
·
Most
common manifestation of GI disease
·
Nausea:
It is a feeling of discomfort in the epigastrium with a conscious desire to
vomit.
·
Vomiting:
It is the forceful ejection of partially digested food and secretions from the
upper GI tract.
Etiology
·
Found
in wide variety of GI as well as unrelated to GI diseases
·
Pregnancy
·
Infectious
diseases - Food poisoning
·
Central
nervous system disorders - Meningitis, CNS tumors, head injury
·
Cardiovascular
problems - Myocardial infarction, Heart failure
·
Metabolic
disorders - Diabetic mellitus, Addison's
disease, uremia
·
Side
effects of drugs - Chemotherapy,
opioids, digitalis
·
Psychological
factors - Stress, fear
·
Motion
sickness,
·
migraines
·
Overeating
·
Appendicitis
·
Inflammation
of the labyrinth
·
Intestinal
obstruction
·
Head
injury
Pathophysiology
·
Vomiting
is believed to be controlled by two distinct brain centers—the vomiting center and the chemoreceptor trigger zone—both located in the medulla oblongata.
The vomiting center initiates and controls the act of emesis, which involves a
series of contractions of the smooth muscles lining the digestive tract.
·
Vomiting
center in brainstem receives input from various stimuli.
·
Neural
impulses reach the vomiting center via afferent pathways through branches of
the autonomic nervous system.
·
Visceral
receptors for these afferent fibers are located in the GI tract, kidneys, heart
& uterus.
·
When
stimulated, these receptors relay information to the vomiting center and
Initiates the vomiting reflex.
·
Chemoreceptor
trigger zone (CTZ) located on the floor of the fourth ventricle in the brain
responds to chemical stimuli of drugs and toxins. Labyrinthine stimulation
(motion sickness)
·
Ones
stimulated, CTZ transmits impulses directly to vomiting center.
·
Vomiting
can also occur when the GI tract becomes overly irritated, excited, or
distended.
·
It
can be a protective mechanism to rid the body of spoiled or irritating foods
and liquids.
Clinical
manifestations
·
Nausea
- a subjective complaint
·
Anorexia
- lack of appetite
·
Dehydration
- if vomiting prolongs
·
Electrolyte
imbalance, Hypotension
·
Weakness,
Weight loss
·
Metabolic
alkalosis - loss of gastric HCl
Management Goal:
·
To
determine and treat the underlying cause.
·
To
provide symptomatic relief.
History:
·
Frequency,
precipitating factors
·
Contents
of vomiting - bile,
·
Color
of emesis- Coffee ground
·
Type
- projectile vomiting, regurgitation
·
Time
- early morning
Medical management
Drug therapy:
Antiemetic
·
Antihistamine:
promethazine (Phenargan)
·
Phenothiazines:
Chlorpromazine
·
Prokinetic:
Domperidone, Metoclopramide(Perinorm)
·
Serotonin
antagonist: Ondansetron
·
Others:
Dexamethasone for cancer chemotherapy induced emesis
Nutrition
therapy:
·
For
severe vomiting - fluid therapy with
electrolyte and GIucose replacement, NG tube aspiration
·
For
improved condition- high carbohydrate and low fat diet. Small and frequent
foods
Nursing Management
Assessment
·
Important
health information: Past health history, medicines, surgery.
·
Physical
examination:
·
General-
Lethargy, sunken eyeball,
·
Integumentary
- Pallor, dry mucus membrane, poor skin turgor
·
GI
- Amount, frequency, character, content,
color of vomitus
·
Urinary
- Decreased output, concentrated urine
Nursing diagnosis
Deficient
fluid volume related to prolonged vomiting and inability to ingest, digest or
absorb food and fluids.
Nurses
responsibilities
·
Assess
duration, frequency and nature of nausea and vomiting.
·
Provide
mouth care, Administer & monitor amount & type of IV fluid
·
Provide
small amounts of clear fluids,
·
Main
I/O chart,
·
Monitor
lab reports
·
Instruct
patient to resume eating cautiously with bland non-irritating foods in small
amounts to avoid irritating the stomach and initiating recurrence of nausea and
vomiting.
·
Nausea
and vomiting are common complaints and anti-emetics may play an important part
in their management, but the primary objective should always be first to
identify the underlying cause and if possible treat that primary disorder.
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